Case #50 – GCC, VEP and RAPDx

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With the patient’s distance prescription in place ( with an additional +1.00 D to compensate for the 1 meter working distance), the central 5 degrees of the visual field is focused onto the central 5 degrees of the fundus which corresponds to the macula. Action potentials are then generated by the ganglion cells which are

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Visual Evoked Potentials (VEPs) With VEPs, the patient is treated as a “black box” and the patient looks at checks (usually black and white) that pattern reverse - white checks to black and black checks to white at a specific reversal rate. The pattern reversal stimulus creates a change across the central retina and this

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Case 1: Zeiss Humphrey 30-2 Visual Fields OU The GCC asymmetry is not reflected in the central threshold visual fields. To learn the basics of GCC. (#RR21)

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Case 1 Optovue iWellness OU Exam The iWellness exam was performed. This test has recently been reported to have 91% sensitivity and specificity.1 Although the SD OCT scans through the fovea were normal in each eye, the Ganglion Cell Complex (GCC) was observably thinner in the right eye than in the left eye. Quantitatively,

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Case 1: Zeiss VISUCAM Color Fundus Image OU Comparison of the right and left maculas and discs of each eye failed to reveal an explanation for her symptoms.

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The pupils, observed by several different clinicians, were judged as normal and a RAPD was not observed. The macula and discs were unremarkable in each eye. Color vision with PIP yielded 11/11 correct in each eye, but the patient commented that the test was more difficult with the right eye.

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Case 1: A 29 year old white female presented complaining of blurred vision in her right eye since having a sinus infection about 4 weeks earlier. The sinus infection resolved but the vision in the right eye was reported as “off” when compared to her left eye. General health history was unremarkable. Best corrected

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Dx of Optic Neuropatihies - Beyond the Basics • Clinically encountered optic nerve disorders encompass glaucoma and a wide range of non-glaucomatous optic neuropathies. • EtiologIes range from congenital and non-progressive disorders such as ON hypoplasia to MS to brain tumors to hereditary and progressive optic neuropathies. • Technology now available to clinicians in

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